Enterococcus faecalis Surface Protein and Urinary Tract Infections
نویسندگان
چکیده
منابع مشابه
Spread of Enterococcal Surface Protein in Antibiotic Resistant Entero-coccus faecium and Enterococcus faecalis isolates from Urinary Tract Infections
Enterococci rank among leading cause of nosocomial bacteremia and urinary tract infection in hospital and community acquired infections. Several traits that may contribute to enhanced virulence have been identified in Enterococci. Extracellular surface protein (Esp) is a virulence factor that contributes in biofilm formation and resistance to environmental stresses. In this study we aimed to de...
متن کاملRole of Enterococcus faecalis surface protein Esp in the pathogenesis of ascending urinary tract infection.
Enterococcus faecalis bacteria isolated from patients with bacteremia, endocarditis, and urinary tract infections more frequently express the surface protein Esp than do fecal isolates. To assess the role of Esp in colonization and persistence of E. faecalis in an animal model of ascending urinary tract infection, we compared an Esp(+) strain of E. faecalis to its isogenic Esp-deficient mutant....
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Enterococcus spp. as pathogens have increased, but the sources of infection often remain unclear. To investigate whether poultry might be a reservoir for E. faecalis-associated urinary tract infections (UTIs) in humans, we characterized E. faecalis isolates from patients in Vietnam with UTIs during January 2008-January 2010 and poultry living in close contact with them by multilocus sequence ty...
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25 26 We examined E. faecalis strains clinically isolated from 100 UTI patients for their 27 susceptibility to levofloxacin (LVX) by measuring the minimal inhibitory concentration 28 (MIC), and investigated amino acid mutations by direct DNA sequencing which was 29 then correlated with LVX resistance. Next we studied risk factors for LVX resistance 30 such as age, sex, and previous fluoroquinol...
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Urinary catheterization elicits major histological and immunological changes that render the bladder susceptible to microbial invasion, colonization, and dissemination. However, it is not understood how catheters induce these changes, how these changes act to promote infection, or whether they may have any protective benefit. In the present study, we examined how catheter-associated inflammatio...
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ژورنال
عنوان ژورنال: Infection Control & Hospital Epidemiology
سال: 2001
ISSN: 0899-823X,1559-6834
DOI: 10.1017/s0195941700066868